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Further, oxidative damage is strongly related with the occurrence of neuroinflammation, enhanced endoplasmic reticulum (ER) stress, excessive apoptosis and impaired autophagy, where these mechanisms are interconnected with the pathophysiology of neurodevelopmental disorders, such as ADHD (Coulson et al

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The direct downstream targets of mTORC1 are ribosomal S6 kinase (p70S6K) and eukaryotic initiation factor 4Ebinding proteins (4EBP) which regulate protein translation, cell size and cell cycle progression 7,8

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